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PI 3-kinase/p110alpha-mediated inactivation of FOXO1 in SMC ameliorates onset and progression of abdominal aortic aneurysms

29 August 2025 (07:00 - 18:00)
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Congress Presentation Part of: Molecular drivers of vascular remodelling: from atherosclerosis to aneurysm therapy Atherosclerosis, Cerebrovascular Diseases, Aneurysm, Restenosis ESC Professional Premium Access ESC Congress 2025 European Society of Cardiology

About the speaker

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Doctor Marius Vantler

Cologne University Hospital - Heart Center, Cologne (Germany)

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PI3Ka protects against arteriosclerosis progression and plaque instability by controlling vascular smooth muscle cell functions
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Dapagliflozin suppresses vascular smooth muscle cell senescence by promoting autophagy
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Evidence from normal human arteries: IRF7 triggers atherosclerosis by enhancing vascular smooth muscle cells inflammation
Doctor S. Cheng (Nanjing, CN)
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Mass spectrometry-based proteomics identifies high glucose-induced changes in vascular smooth muscle (VSMCs) and sustained extracellular signal-regulated kinases (ERK) activation
Mrs R. Aldahhasi (Leicester, GB)
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Access the full session

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Molecular drivers of vascular remodelling: from atherosclerosis to aneurysm therapy

The Event

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ESC Congress 2025

29 August - 1 September 2025
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PI 3-kinase/AKT/FOXO1 signaling in smooth muscle cells protects against abdominal aortic aneurysm
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Congress Presentation
Importance of smooth muscle p110alpha/AKT/FOXO1 signaling for the development of abdominal aortic aneurysm

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