Profile picture

Doctor Marius Vantler

Cologne University Hospital - Heart Center, Cologne (Germany)
Membership: ESC Professional Member
Follow
Logo ESC

Contributor content

PI 3-kinase/p110alpha-mediated inactivation of FOXO1 in SMC ameliorates onset and progression of abdominal aortic aneurysms
Presentation
PI 3-kinase/p110alpha-mediated inactivation of FOXO1 in SMC ameliorates onset and progression of abdominal aortic aneurysms
PI 3-kinase/AKT/FOXO1 signaling in smooth muscle cells protects against abdominal aortic aneurysm
Presentation
PI 3-kinase/AKT/FOXO1 signaling in smooth muscle cells protects against abdominal aortic aneurysm
Importance of smooth muscle p110alpha/AKT/FOXO1 signaling for the development of abdominal aortic aneurysm
Presentation
Importance of smooth muscle p110alpha/AKT/FOXO1 signaling for the development of abdominal aortic aneurysm
Lack of PI 3-kinase isoform p110alpha impairs SMC differentiation and proliferation and promotes aortic aneurysm formation
Presentation
Lack of PI 3-kinase isoform p110alpha impairs SMC differentiation and proliferation and promotes aortic aneurysm formation
PI 3-kinase isoform PI3Kalpha controls smooth muscle cell functionality and protects against aortic aneurysm formation
Presentation
PI 3-kinase isoform PI3Kalpha controls smooth muscle cell functionality and protects against aortic aneurysm formation
Lack of PI 3-kinase isoform p110alpha in smooth muscle cells impairs aortic wall homoeostasis and thus promotes aortic aneurysm formation
Presentation
Lack of PI 3-kinase isoform p110alpha in smooth muscle cells impairs aortic wall homoeostasis and thus promotes aortic aneurysm formation
Deficiency of PI 3-kinase isoform p110alpha in smooth muscle cells impairs vascular integrity and promotes aortic aneurysm formation
Presentation
Deficiency of PI 3-kinase isoform p110alpha in smooth muscle cells impairs vascular integrity and promotes aortic aneurysm formation
SGC-stimulation via BAY 41-2272 exerts antiatherogenic effects in LDLR- and ApoE-deficient mice
Presentation
SGC-stimulation via BAY 41-2272 exerts antiatherogenic effects in LDLR- and ApoE-deficient mice

ESC 365 is supported by