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Conditional over-expression of heme-oxygenase 1 in myelomonocytic cells attenuates vascular inflammation and prevents the development of heart failure in mice with excess neurohormonal activation

31 August 2025 (08:55 - 09:05)
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Congress Presentation Part of: Biomarkers and vascular mechanisms of hypertension and its organ damage Renin-Angiotensin System ESC Professional Premium Access ESC Congress 2025 European Society of Cardiology

About the speaker

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Professor Philip Wenzel

Johannes Gutenberg University Mainz (JGU), Mainz (Germany)

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High-resolution transcriptomic profiling of the aortic cellular landscape during hypertension reveals novel drivers of vascular fibrosis
Professor G. Drummond (Bundoora, AU)
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Shear stress-induced endothelial HEG1 signaling regulates vascular tone and blood pressure
Mr W. Wu (Shanghai, CN)
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RNF40-mediated Parkin ubiquitination exacerbates blood-brain barrier disruption in hypertension
Doctor X. Zhao (Lanzhou, CN)
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Hypertension mediated organ damage in relation to the severity of erectile dysfunction, the endogenous androgen levels and central vascular stiffness in middle aged men with essential hypertension
Doctor I. Dimitroglou (Athens, GR)
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Biomarkers and vascular mechanisms of hypertension and its organ damage
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The Event

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ESC Congress 2025

31 August 2025 08:55 CET
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Conditional over-expression of heme-oxygenase 1 in myelomonocytic cells reduces AngII-induced hypertension and vascular inflammation in mice
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