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SMAD3 silencing induces Epithelial-to-Mesenchymal Transition (EMT) towards cardiac pericyte progenitors with proangiogenic potential in hiPSC-derived epicardial cells

30 August 2024 (12:00 - 12:50)
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Congress Presentation Part of: Targeting endothelial function Stem Cells, Cell Cycle, Cell Senescence, Cell Death ESC Professional Premium Access ESC Congress 2024 European Society of Cardiology

About the speaker

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Doctor Yutaro Miyoshi

Kyoto University Graduate School of Medicine, Kyoto (Japan)

8 More presentations in this session

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Increased endothelial cell uptake of erythrocyte-derived extracellular vesicles carrying arginase-1 induces endothelial dysfunction in type 2 diabetes
Doctor A. Collado (Stockholm, SE)
Abstract Slides
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Blockade of endothelial to mesenchymal transition (EndMT) by an inhibitor of histone methyltransferase EZH2 attenuates atherosclerosis in diabetes
Doctor A. Khan (Melbourne, AU)
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Inhibition of ketohexokinase prevents fructose-induced insulin resistance and endothelial dysfunction in high fat and sucrose-fed mice via enhancing energy expenditure
Doctor H. Viswambharan (Leeds, GB)
Abstract Slides Video
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Restoration of autophagy reduces diabetes-induced endothelial dysfunction
Doctor L. D'Ambrosio (Venafro, IT)
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STING-mediated DNA damage response in diabetes-induced vascular complications
Ms O. Bavuu (Osaka, JP)
Abstract Slides

Access the full session

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Targeting endothelial function

The Event

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ESC Congress 2024

30 August 2024 12:00 CET
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ESC 365 is supported by

novartis Explore sponsored resources