ESC 365 - Disease-associated calmodulin mutations disrupt L-type Ca2+ channel (Cav1.2) activity and CaMKIIdelta phosphorylation in long QT syndrome

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Disease-associated calmodulin mutations disrupt L-type Ca2+ channel (Cav1.2) activity and CaMKIIdelta phosphorylation in long QT syndrome

Session: Mechanisms of ventricular arrhythmias

Topic: Arrhythmias

Speaker: Doctor N. Helassa (Liverpool, GB)

Event: ESC Congress 2022

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Doctor Nordine Helassa

University of Liverpool, Liverpool (United Kingdom of Great Britain & Northern Ireland)

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7 more presentations in this session

Synapsin 2 regulates NCX1 trafficking and is down-regulated in the failing myocardium, which increases the risk of ventricular arrhythmia and heart failure mortality

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Empagliflozin attenuates arrhythmogenesis via inhibition of O-GlcNAcylation in diastolic phase of diabetic cardiomyopathy.

Speaker: Doctor T. Kadosaka (Sapporo, JP)

Long QT syndrome-associated calmodulin mutations and their interactions with the Kv7.1 voltage-gated potassium channel

Speaker: Mr L. McCormick (Leeds, GB)

Utility of in vivo zebrafish cardiac assay to predict the functional impact of KCNQ1 variants

Speaker: Mr S. Cui (Kanazawa, JP)

Acute afterload leads to increased electrophysiological heterogeneity after myocardial infarction.

Speaker: Doctor S. Ingelaere (Leuven, BE)

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Mechanisms of ventricular arrhythmias

Speakers: Doctor N. Helassa, Doctor A. Ottesen, Doctor T. Kadosaka, Mr L. McCormick, Mr S. Cui...

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ESC Congress 2022

26 August - 29 August 2022

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