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The ketone body 3-hydroxybutyrate increases cardiac output through afterload reduction and not through increased left ventricular contractility

21 May 2022 (09:40 - 10:25)
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Congress Presentation Part of: From mechanisms to targets over the heart failure spectrum Heart Failure HFA Premium Access Heart Failure 2022

About the speaker

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Doctor Nigopan Gopalasingam

Goedstrup Hospital, Herning (Denmark)

5 More presentations in this session

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ATPIF-1 regulates the cardiac mitochondrial permeability transition pore and mitochondrial shape in mice
Mr P. Sanchez (Groningen, NL)
Abstract Slides
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Pirfenidone added to standard therapy for myocardial infarction further reduces left ventricular fibrosis: a preclinical study
Doctor A. Aimo (Pisa, IT)
Abstract Slides
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Effect of different post-myocardial infarction exercise training modality on cardiac calcium sensitivity and function
Professor J. Zhang (San Antonio, US)
Abstract Slides
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Endoplasmic reticulum stress as a therapeutic target for the treatment of cardiac and renal damage post myocardial infarction in the context of obesity
Miss B. Delgado Valero (Madrid, ES)
Abstract Slides
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Pressure overload-induced cardiac hypertrophy stimulates tumour growth in tumour prone apc-min mice
Ms S. De Wit (Groningen, NL)
Abstract Slides

Access the full session

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From mechanisms to targets over the heart failure spectrum

The Event

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Heart Failure 2022

21 May 2022 09:40 CET
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ESC 365 is supported by

novartis Explore sponsored resources
novo-nordisk Explore sponsored resources