Acute effects of a mavacamten-like myosin-inhibitor (MYK-581 in a feline model of obstructed hypertrophic cardiomyopathy: evidence of improved ventricular filling (beyond obstruction reprieve)
Disruption of the architecture of the junctional sarcoplasmic reticulum in recessive catecholaminergic polymorphic ventricular tachycardia is caused by the er-shaping proteins reep5 and climp63