Non-transcriptional disruption of Ca2+i homeostasis and Cx43 function in the right ventricle precedes overt arrhythmogenic cardiomyopathy in PKP2-deficient mice.

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Non-transcriptional disruption of Ca2+i homeostasis and Cx43 function in the right ventricle precedes overt arrhythmogenic cardiomyopathy in PKP2-deficient mice.

Session: Insights into arrhythmia mechanism and therapies

Topic: Ion Channels, Electrophysiology

Speaker: Associate Professor M. Cerrone (New York, US)

Event: ESC Congress 2019

Congress Presentation

About the speaker

Associate Professor Marina Cerrone

New York University Langone Medical Center, New York (United States of America)

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5 more presentations in this session

Loss-of-spen disturbs cardiac conduction in zebrafish mediated by impaired connexin43 function.

Speaker: Doctor M. Rattka (Essen, DE)

Targeting INaL by a neuronal sodium channel isoform improves survival in a CaMKII-transgenic heart failure mouse model

Speaker: Mr P. Bengel (Goettingen, DE)

Effects of atrial fibrillation on the human ventricle

Speaker: Doctor S. Pabel (Regensburg, DE)

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Abstract

Increased Gao expression underlies cardiac dysfunction and lethal arrhythmias accompanied with abnormal Ca2+ handling

Speaker: Doctor H. Inazumi (Kyoto, JP)

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Abstract

Enhanced gap junction coupling organised and terminated acute ventricular fibrillation in ex-vivo perfused hearts

Speaker: Doctor B. Handa (London, GB)

Access the full session

Insights into arrhythmia mechanism and therapies

Speakers: Associate Professor M. Cerrone, Doctor M. Rattka, Mr P. Bengel, Doctor S. Pabel, Doctor H. Inazumi...

Access

About the event

ESC Congress 2019

31 August - 4 September 2019

Sessions Presentations

Congress

Non-transcriptional disruption of Ca2+i homeostasis and Cx43 function in the right ventricle precedes overt arrhythmogenic cardiomyopathy in PKP2-deficient mice.

About the speaker

Associate Professor Marina Cerrone

5 more presentations in this session

Loss-of-spen disturbs cardiac conduction in zebrafish mediated by impaired connexin43 function.

Targeting INaL by a neuronal sodium channel isoform improves survival in a CaMKII-transgenic heart failure mouse model

Effects of atrial fibrillation on the human ventricle

Increased Gao expression underlies cardiac dysfunction and lethal arrhythmias accompanied with abnormal Ca2+ handling

Enhanced gap junction coupling organised and terminated acute ventricular fibrillation in ex-vivo perfused hearts

Access the full session

Insights into arrhythmia mechanism and therapies

About the event

ESC Congress 2019

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