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The incretin hormone GIP is upregulated in patients with atherosclerosis and stabilizes plaques in ApoE-/- mice by blocking proinflammatory signaling in macrophages

27 August 2018 (14:47 - 15:01)
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Congress Presentation Part of: Inflammation as a critical component of atherosclerosis Atherosclerosis, Cerebrovascular Diseases, Aneurysm, Restenosis ESC Premium Access ESC Congress 2018

About the speaker

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Mr Robert Werner Mertens

RWTH University Hospital Aachen, Aachen (Germany)

4 More presentations in this session

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Inflammation as a critical component of atherosclerosis - State of the art
Professor P. Libby (Boston, US)
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Activation of canonical proinflammatory pathways in smooth muscle cells exerts paradoxical atheroprotective effects
Doctor K. Stark (Munich, DE)
Abstract
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Stat4 deficiency exacerbates atherosclerosis through promoting foam cell formation via pi3k/akt/nfkb/mir-9/acat1 pathway in ApoE-/- mice
Doctor L. Xu (Shanghai, CN)
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Inflammation and the vulnerable plaque.
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Inflammation as a critical component of atherosclerosis

The Event

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ESC Congress 2018

27 August 2018 14:47 CET

ESC 365 is supported by

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ESC 365 is supported by

novartis Explore sponsored resources
novo-nordisk Explore sponsored resources