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Excessive mitochondrial reactive oxygen species emission from circulating blood cells is associated with severity of heart failure and exercise intolerance

27 August 2018 (08:45 - 09:00)
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Congress Presentation Part of: Heart Failure: new targets for treatment Heart Failure ESC Premium Access ESC Congress 2018

About the speaker

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Doctor Ryosuke Shirakawa

Hokkaido University, Sapporo (Japan)

5 More presentations in this session

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Angiotensin II-dependent activation of NADPH oxidase 4 contributes to muscle wasting in mice via downregulation of NF-E2-related factor 2
Doctor T. Kadoguchi (Tokyo, JP)
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Positive cardiac inotropy by carvedilol via unique beta-arrestin2-dependent SERCA2a stimulation
Associate Professor A. Lymperopoulos (Fort Lauderdale, US)
Abstract Slides Video
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Empagliflozin potently reduces sarcoplasmic Ca leak and increases Ca transient amplitude of human failing ventricular cardiomyocytes
Mr J. Mustroph (Regensburg, DE)
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HECTD3 attenuates cardiac hypertrophy acting as an E3 ligase of SUMO2
Doctor A. Rangrez (Kiel, DE)
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RNA methylation in cardiac hypertrophy and heart failure
Mr E. Buchholz (Goettingen, DE)
Abstract

Access the full session

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Heart Failure: new targets for treatment

The Event

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ESC Congress 2018

27 August 2018 08:45 CET
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