Design of models for predicting the development of heart failure with reduced ejection fraction and preserved ejection fraction in patients with acquired heart diseases in the postoperative period
Hypertrophic cardiomyopathy: variable expression of mutant and wildtype protein from cell-to-cell leading to contractile imbalance - a possible trigger for pathogenesis
Shock wave therapy improves cardiac function in a model of chronic ischemic heart failure-evidence for a mechanism involving VEGF signaling and the extracellular matrix
Speaker:
Associate Professor J. Holfeld (Innsbruck, AT)
Non-hypertrophic and hypertrophic human cardiomyocytes secrete unprocessed ANP and BNP precursors (proANP/proBNP) and exhibit a similar efficiency in their processing