Changes in titin phosphorylation and PKG activity underlie diastolic dysfunction at the cross-bridge level in the early stages of type 2 diabetes

26 May 2018 (00:00 - 00:00)

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About the speaker

Doctor Mark Waddingham

National Cerebral & Cardiovascular Center, Suita (Japan)

5 More presentations in this session

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Generation and validation of a mouse model of the phospholamban p.Arg14del mutation

Mr T. Eijgenraam (Groningen, NL)

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HDAC inhibitors repress cardiac hypertrophy through hyperacetylation of 14-3-3 chaperones and nuclear accumulation of class II HDACs

Mr D. Finke (Heidelberg, DE)

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Myocyte BDNF generation prevents chronic post-ischemic decompensation via cardiac reinnervation: the role of beta-AR and GRK2 signals.

Doctor A. Cannavo (Napoly, IT)

Abstract Slides Video

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Desmosomal integrity is challenged by exercise-induced strain in iPSC-derived cardiomyocytes with a novel desmoplakin mutation

Ms M. Vermeer (Groningen, NL)

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The myocardial infarction context dictates the phenotypic polarisation of antigen-specific CD4+T-cells and favours pro-healing responses in the wounded myocardium

Mr M. Rieckmann (Halle, DE)

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Young Investigator Award: Basic Science and Translational Science

The Event

Heart Failure 2018

26 May - 29 May 2018

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