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Changes in titin phosphorylation and PKG activity underlie diastolic dysfunction at the cross-bridge level in the early stages of type 2 diabetes

27 May 2018 (14:45 - 15:00)
Organised by:
Congress Presentation Part of: Young Investigator Award: Basic Science and Translational Science Cardiac Biology and Physiology HFA Premium Access Heart Failure 2018

About the speaker

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Doctor Mark Waddingham

National Cerebral & Cardiovascular Center, Suita (Japan)

5 More presentations in this session

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Generation and validation of a mouse model of the phospholamban p.Arg14del mutation
Mr T. Eijgenraam (Groningen, NL)
Abstract
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HDAC inhibitors repress cardiac hypertrophy through hyperacetylation of 14-3-3 chaperones and nuclear accumulation of class II HDACs
Mr D. Finke (Heidelberg, DE)
Abstract
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Myocyte BDNF generation prevents chronic post-ischemic decompensation via cardiac reinnervation: the role of beta-AR and GRK2 signals.
Doctor A. Cannavo (Napoly, IT)
Abstract Slides Video
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Desmosomal integrity is challenged by exercise-induced strain in iPSC-derived cardiomyocytes with a novel desmoplakin mutation
Ms M. Vermeer (Groningen, NL)
Abstract
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The myocardial infarction context dictates the phenotypic polarisation of antigen-specific CD4+T-cells and favours pro-healing responses in the wounded myocardium
Mr M. Rieckmann (Halle, DE)
Abstract

Access the full session

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Young Investigator Award: Basic Science and Translational Science

The Event

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Heart Failure 2018

27 May 2018 14:45 CET
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novartis Explore sponsored resources