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Epac1-mediated RE/mitochondria interactions promotes mitochondrial Ca2+ overload and cell death during ischemia/reperfusion injury

29 April 2017 (08:30 - 08:36)
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Congress Presentation Part of: Rapid Fire 1 - Basic Science Metabolism / Ischemia / Reperfusion / Preconditioning HFA Premium Access Heart Failure 2017 - 4th World Congress on Acute Heart Failure

About the speaker

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Doctor Loubina Fazal

Inserm, Paris (France)

13 More presentations in this session

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Intramyocardial delivery of mesenchymal stem cells transfected with minicircle-HIF-1a decreases LV adverse remodelling via release of cardioprotective miRNAs and pro-angiogenic factors
Doctor E. Gara (Budapest, HU)
Abstract
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Prophylactic combined heart failure treatment attenuates the development of doxorubicin induced systolic and diastolic dysfunction in rats
Ms M. Lodi (Debrecen, HU)
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NPA7, an innovative multivalent designer natriuretic peptide: Co-therapy with Furosemide to mediate renoprotection in experimental ADHF
Doctor L. Meems (Groningen, NL)
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Eosinophils have an essential role in promoting a type 2 innate immune response and cardiac repair following myocardial infarction.
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NADPH oxidase-4 promotes adaptive cardiac remodelling in the chronically stressed heart by driving protein O-GlcNAcylation and enhancing cardiac fatty acid oxidation
Doctor A. Nabeebaccus (London, GB)
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Access the full session

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Rapid Fire 1 - Basic Science

The Event

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Heart Failure 2017 - 4th World Congress on Acute Heart Failure

29 April 2017 08:30 CET

ESC 365 is supported by

novartis Explore sponsored resources

ESC 365 is supported by

novartis Explore sponsored resources