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Myeloid cell specific alpha1-AMPK deletion induces a proinflammatory phenotype and enhances angiotensin II-mediated vascular dysfunction

29 August 2016 (15:35 - 15:42)
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Congress Presentation Part of: Vascular biology Endothelial function ESC Premium Access ESC Congress 2016

About the speaker

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Doctor Thomas Jansen

Johannes Gutenberg University Mainz (JGU), Mainz (Germany)

6 More presentations in this session

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Glycemic control with ipragliflozin, a novel selective SGLT2 inhibitor, ameliorated endothelial dysfunction in obese and non-obese diabetic mice.
Miss H. Salim (Surabaya, ID)
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Endothelial PGC-1alpha preserves endothelial function during chronic angiotensin II treatment by maintaining eNOS expression, reducing vascular oxidative stress and supporting the antioxidant defense
Doctor T. Jansen (Mainz, DE)
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Impact of the adiponectin paralog CTRP13 on endothelial cells
Doctor M. Aslam (Giessen, DE)
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Monocyte-platelet aggregates associated with CD14++CD16+ monocytes predict diffuse coronary artery disease: relationship to microvascular function
Doctor R. Brown (Newcastle-Upon-Tyne, GB)
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A direct thrombin inhibitor paradoxically enhances thrombin generation via suppression of protein C activation and factor Va degradation in human plasma
Doctor Y. Morishima (Tokyo, JP)
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Access the full session

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Vascular biology

The Event

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ESC Congress 2016

29 August 2016 15:35 CET

ESC 365 is supported by

novartis Explore sponsored resources

ESC 365 is supported by

novartis Explore sponsored resources