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HFWM: PKA/PKC mediated changes in phosphorylation of inhibitor-1 at residues thr35 and ser67 contribute to elevated intracellular sodium in failing hearts

21 May 2016 (00:00 - 00:00)
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Congress Presentation Part of: Poster Session 3 - Basic Science: Cell signalling Cell Signalling HFA Premium Access HEART FAILURE 2016

About the speaker

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Doctor Davor Pavlovic

University of Birmingham, Birmingham (United Kingdom of Great Britain & Northern Ireland)

5 More presentations in this session

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HFWM: A new potential strategy for targeting pathological cardiac hypertrophy: the dimer interface of ERK1/2
Professor K. Lorenz (Wuerzburg, DE)
Abstract
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HFWM: A central role for Hippo pathway and actin filament dynamics in microRNA-induced cardiomyocyte proliferation
Miss C. Torrini (Trieste, IT)
Abstract
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HFWM: Cardiac specific overexpression of phosphodiesterase 4b in mice limits beta-adrenergic response and associated pro-arrhythmic ca(2+) events
Doctor S. Karam (Chatenay-Malabry, FR)
Abstract
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Evidence and role of neuronal sodium channel Nav1.8 in human failing cardiomyocytes
Doctor S. Ahmad (Goettingen, DE)
Abstract Slides
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Impact of GMP-PKG pathway modulation on titin phosphorylation and titin-based myocardial passive stiffness
Professor N. Hamdani (Bochum, DE)

Access the full session

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Poster Session 3 - Basic Science: Cell signalling

The Event

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HEART FAILURE 2016

21 May - 24 May 2016

ESC 365 is supported by

novartis Explore sponsored resources

ESC 365 is supported by

novartis Explore sponsored resources