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Doctor Walaa Fakih

Strasbourg (France)
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Biography
Researcher in Translational Cardiovascular Medicine and Pharmacotherapy
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Contributor content

Epicardial adipose tissue-derived adipo-cytokines induce pro-oxidant and pro-remodeling responses in atrial endothelial cells: synergic inhibitory effects of SGLT2 inhibition and GLP-1R activation
Presentation
Epicardial adipose tissue-derived adipo-cytokines induce pro-oxidant and pro-remodeling responses in atrial endothelial cells: synergic inhibitory effects of SGLT2 inhibition and GLP-1R activation
Inflamed atrial tissues of patients with atrial fibrillation are characterized by pro-oxidant and pro-remodeling responses: synergistic inhibition by an SGLT2 inhibitor plus a GLP-1R activation
Presentation
Inflamed atrial tissues of patients with atrial fibrillation are characterized by pro-oxidant and pro-remodeling responses: synergistic inhibition by an SGLT2 inhibitor plus a GLP-1R activation
Human epicardial adipose tissue-derived factors promote atrial endothelial dysfunction: role of pro-inflammatory cytokines and AT1R/NADPH oxidases/SGLT2 pro-oxidant pathway
Presentation
Human epicardial adipose tissue-derived factors promote atrial endothelial dysfunction: role of pro-inflammatory cytokines and AT1R/NADPH oxidases/SGLT2 pro-oxidant pathway
TNF-alfa promotes NF-kB-dependent oxidative stress in human left appendages and in atrial endothelial cells: Role of SGLT2
Presentation
TNF-alfa promotes NF-kB-dependent oxidative stress in human left appendages and in atrial endothelial cells: Role of SGLT2
Increased pro-remodeling and pro-thrombotic responses in the left compared to the right atrial appendage: role of low-grade inflammation and the AT1R/NADPH oxidases/SGLT2 pro-oxidant pathway
Presentation
Increased pro-remodeling and pro-thrombotic responses in the left compared to the right atrial appendage: role of low-grade inflammation and the AT1R/NADPH oxidases/SGLT2 pro-oxidant pathway
Upregulation of SGLT1 and 2 promotes oxidative stress in right atrial appendages of patients with low-grade inflammatory responses: potential role in atrial fibrillation
Presentation
Upregulation of SGLT1 and 2 promotes oxidative stress in right atrial appendages of patients with low-grade inflammatory responses: potential role in atrial fibrillation
FXa promotes endothelial dysfunction and pro-remodeling responses in atrial endothelial cells involving PAR1 and 2 and AT1R/NADPH oxidase/SGLT1/2 pro-oxidant pathways
Presentation
FXa promotes endothelial dysfunction and pro-remodeling responses in atrial endothelial cells involving PAR1 and 2 and AT1R/NADPH oxidase/SGLT1/2 pro-oxidant pathways
Thrombin and factor Xa promote premature senescence and remodeling in areas at risk of the left appendage and in atrial endothelial cells: Potential modulation by SGLT1 and/or SGLT2 inhibition
Presentation
Thrombin and factor Xa promote premature senescence and remodeling in areas at risk of the left appendage and in atrial endothelial cells: Potential modulation by SGLT1 and/or SGLT2 inhibition

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