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Doctor Jochen Dutzmann

University Hospital Halle (Saale), Halle (Germany)
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Biography
Jochen Dutzmann gained his MD from the Justus Liebig University in Giessen, Germany in 2013. During his studies, he spent several months as a visiting student at The Children's Hospital of the University of Colorado in Denver, as a research fellow at the Cardiovascular Division of the Brigham and Women's Hospital/Harvard Medical School in Boston, USA as well as a MD student at the Dept. of Internal Medicine of the University Hospital Basel, Switzerland. He served as a clinical resident and fellow at the Dept. of Cardiology and Vascular Medicine of the Hannover Medical School, Germany and became a specialist for emergency medicine in 2018 and for internal medicine in 2019. In 2019, he started working at the Division of Cardiology, Vascular Medicine and Internal Intensive Care of the Mid-German Heart Center in Halle, Germany as an attending physician and clinician scientist. His research foci are vascular remodeling, acute cardiac care, palliative cardiac care, and medical ethics.
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Empagliflozin impairs smooth muscle cell proliferation, accelerates endothelial regeneration, and prevents neointima formation by altered expression of the vasoactive peptide apelin
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Empagliflozin impairs smooth muscle cell proliferation, accelerates endothelial regeneration, and prevents neointima formation by altered expression of the vasoactive peptide apelin
IL-6 production in the vascular adventitia and adventitia-media-crosstalk in neointima formation
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IL-6 production in the vascular adventitia and adventitia-media-crosstalk in neointima formation
Adventitial interleukin-6 release is critical for neointima formation
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Adventitial interleukin-6 release is critical for neointima formation
BET bromodomain inhibition permits FoxO1-dependent cell cycle arrest in smooth muscle cells and prevents neointima formation
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BET bromodomain inhibition permits FoxO1-dependent cell cycle arrest in smooth muscle cells and prevents neointima formation
Adventitial interleukin-6 release is critical for neointima formation
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Adventitial interleukin-6 release is critical for neointima formation
Adventitial cytokine production is critical for negative vascular remodelling
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Adventitial cytokine production is critical for negative vascular remodelling
Adventitial activation is critical for negative vascular remodeling
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Adventitial activation is critical for negative vascular remodeling

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