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Associate Professor Attila Kiss

Medical University of Vienna, Vienna (Austria)
Membership: FESC Member
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Biography
Cardiovascular physiology, pathophysiology and pharmacology; experimental models of myocardial ischemia/reperfusion and infarction. Focus on the endogenous adaptation of the heart against ischemic and reperfusion injury (e.g remote conditioning). Importance of altered microRNAs expression and signaling pathways in the development of myocardial ischemia/reperfusion injury and adverse left ventricle remodeling. Novel approaches for optimizing cardioprotection in cardiac surgery. The pathophysiological role of Tenascin C in cardiovascular disorders.
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Contributor content

Cardiovascular phenotype of the duchenne muscular dystrophy carrier female rat
Presentation
Cardiovascular phenotype of the duchenne muscular dystrophy carrier female rat
Increased Tenascin-C expression contributes to cardiac dysfunction and fibrosis in Duchenne muscular dystrophy
Presentation
Increased Tenascin-C expression contributes to cardiac dysfunction and fibrosis in Duchenne muscular dystrophy
Two in one: Neuregulin 1 improves cardiac diastolic and kindney funtcion in chronic kidney disease in rats
Presentation
Two in one: Neuregulin 1 improves cardiac diastolic and kindney funtcion in chronic kidney disease in rats
Beneficial effects of repeated remote ischemic conditioning on left ventricular function following chronic myocardial infarction in rats
Presentation
Beneficial effects of repeated remote ischemic conditioning on left ventricular function following chronic myocardial infarction in rats
Cardioprotection during cardiac surgery: impact of temperature of cardioplegic solution on microRNA profile in a pig model of cardiopulmonary bypass
Presentation
Cardioprotection during cardiac surgery: impact of temperature of cardioplegic solution on microRNA profile in a pig model of cardiopulmonary bypass
Characterization of early left ventricle dysfunction in a relevant experimental  model for human rheumatoid arthritis.
Presentation
Characterization of early left ventricle dysfunction in a relevant experimental model for human rheumatoid arthritis.
Attenuation of myocardial and vascular arginase activity by vagal nerve stimulation via a mechanism involving alpha-7 nicotinic receptor during cardiac ischemia and reperfusion
Presentation
Attenuation of myocardial and vascular arginase activity by vagal nerve stimulation via a mechanism involving alpha-7 nicotinic receptor during cardiac ischemia and reperfusion

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