Bioinformatic analysis of a plakophilin-2-dependent transcription network: implications for the mechanisms of arrhythmogenic right ventricular cardiomyopathy in humans and in boxer dogs
Multilevel analyses of <em>SCN5A</em> mutations in arrhythmogenic
right ventricular dysplasia/cardiomyopathy suggest non-canonical mechanisms for disease
pathogenesis
Super-resolution imaging reveals that loss of the C-terminus of connexin43 limits microtubule plus-end capture and Na<sub>V</sub>1.5 localization at the intercalated disc
