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Doctor Shengnan Li

Zhongda Hospital, Southeast University, Nanjing (China)
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TMEM65 ameliorates post-myocardial infarction heart failure by maintaining mitochondrial calcium homeostasis via inhibiting NCLX ubiquitination
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TMEM65 ameliorates post-myocardial infarction heart failure by maintaining mitochondrial calcium homeostasis via inhibiting NCLX ubiquitination
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DAMPs exacerbate post-myocardial infarction cardiac injury via suppression of AP-1 signaling: critical role of impaired lymphatic endothelial cell proliferation
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DAMPs exacerbate post-myocardial infarction cardiac injury via suppression of AP-1 signaling: critical role of impaired lymphatic endothelial cell proliferation
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