Back

Sympathetic nervous system hyperactivity-induced oxidative stress promoting endothelial dysfunction is dependent on the NADPH oxidases/SGLT2 crosstalk: potential role in cardiogenic shock

29 August 2022 (14:15 - 15:00)
Organised by:
Congress Presentation Part of: Cell-cell interactions Heart Failure ESC Professional Premium Access ESC Congress 2022

About the speaker

Photo
Ms Sophie Louise Kerth

Munich (Germany)

6 More presentations in this session

Presentation thumbnail
Human, mouse scRNA-seq and flow cytometry data in ischemic heart failure models predict immune cell activation signatures with altered endothelial cell cross talk
Mr M. Merten (Frankfurt, DE)
Abstract Slides
Presentation thumbnail
The long non-coding RNA Heat4 is elevated in heart failure patients and mediates anti-inflammatory functions thereby promoting vascular regeneration
Ms J. Kneuer (Leipzig, DE)
Abstract Slides
Presentation thumbnail
The interaction of receptor-type protein tyrosine phosphatases sigma and zeta1 controls cardiac fibroblast migration
Doctor L. Hasselbach (Oldenburg, DE)
Abstract Slides
Presentation thumbnail
Chronic heart failure as a sequel of severe burn injury: first insight into a novel pathological heart-skin axis
Doctor G. Hundeshagen (Heidelberg, DE)
Abstract Slides
Presentation thumbnail
Decrease of RNA editing in the failing heart leads to induction of circRNAs
Miss K. Kokot (Leipzig, DE)
Abstract Slides

Access the full session

Session thumbnail
Cell-cell interactions

The Event

Event poster

ESC Congress 2022

29 August 2022 14:15 CET
You may be interested in
thumbnail
Congress Presentation
Catecholamines induce endothelial dysfunction to promote pro-inflammatory and pro-thrombotic responses: role of the NADPH oxidases/SGLT2 pro-oxidant pathway
thumbnail
Congress Presentation
Takotsubo syndrome induced oxidative stress and endothelial dysfunction in left ventricle of rat model: association with NADPH oxidases/SGLT2 pro- oxidant pathway
thumbnail
Congress Presentation
Patient vascular model reveals anti-inflammatory effects of SGLT2 inhibitor by modulating endothelial-T cell crosstalk

ESC 365 is supported by

novartis Explore sponsored resources
novo-nordisk Explore sponsored resources

ESC 365 is supported by

novartis Explore sponsored resources
novo-nordisk Explore sponsored resources