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Reducing NOX2-derived reactive oxygen species enhances endothelial cell migration and vascular repair in the context of endothelium-specific insulin resistance

29 August 2016 (16:48 - 16:57)
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Congress Presentation Part of: Endothelial cells Vascular biology, other ESC Premium Access ESC Congress 2016

About the speaker

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Doctor Noman Ali

University of Leeds, Leeds (United Kingdom of Great Britain & Northern Ireland)

8 More presentations in this session

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Oxidative stress-induced miR-200c disrupts the regulatory loop among SIRT1, FOXO1 and eNOS, causing endothelial dysfunction
Doctor A. Magenta (Rome, IT)
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Improved endothelial function in translgutaminase-2 knockout mice treated with angiotensin II
Associate Professor C. Savoia (Rome, IT)
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The microRNA miR-483-3p affects vascular response to injury by promoting endothelial cell apoptosis
Doctor K. Kuschnerus (Berlin, DE)
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Absence of endothelial tumor suppressor p53 in mice prevents the enhanced venous thrombus formation and delayed thrombus resolution observed with age.
Professor K. Schaefer (Mainz, DE)
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Deficiency of protease-activated receptor 1 and 2 reduce the progression of experimental pulmonary hypertension in mice
Ms C. Joseph (Cologne, DE)
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Endothelial cells

The Event

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ESC Congress 2016

29 August 2016 16:48 CET

ESC 365 is supported by

novartis Explore sponsored resources

ESC 365 is supported by

novartis Explore sponsored resources