Speaker illustration

Associate Professor Narcisa Tribulova

Centrum of Experimental Medicine, SAS, Bratislava (Slovakia)

Age-related abnormalities in myocardial connexin43 expression, intercellular coupling and ultrastructure may facilitate heart dysfunction.

Event: Heart Failure 2018

Topic: Ventricular Remodelling

Session: Chronic Heart Failure – Pathophysiology and Mechanisms


Up-regulation of myocardial connexin-43 is involved in compensatory response of the heart to acute injury.

Event: Frontiers in CardioVascular Biology 2018

Topic: Signal Transduction, Mechanotransduction

Session: Basic Science - Cardiac Biology and Physiology


Cardiac connexin43 modulates susceptibility of the heart to life-threatening arrhythmias in various experimental models.


Topic: Mechanism (Basic Science in Arrhythmias)

Session: Rapid Fire session 7 - Arrhythmogenesis and antiarrhythmia in experimental models


Radiation-induced myocardial connexin-43 and miR-1 alterations are partially prevented by concomitant treatment with Aspirin and Atorvastatin.

Event: EuroPrevent 2017

Topic: Risk factors and risk prediction

Session: Risk factors: others_Part 2


Treatment with aspirin and atorvastatin attenuate cardiac injury induced by rat chest irradiation: Implication of myocardial miR-1, miR-21, connexin-43 and PKC

Event: Frontiers in CardioVascular Biology 2016

Topic: Genetics, Epigenetics and Genomics

Session: Genetics and epigenetics


Ultrastructural alterations reflecting Ca2+ handling and cell-to-cell coupling disorders precede occurrence of severe arrhythmias in intact animal heart.

Event: Frontiers in CardioVascular Biology 2016

Topic: Cell death / apoptosis / autophagy - Heart

Session: Cell death and apoptosis - Heart


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