Mrs Julia Moellmann

RWTH University Hospital Aachen, Aachen (Germany)

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The SGLT2 inhibitor ertugliflozin causes a switch of cardiac substrate utilization leading to reduced cardiac mTOR-signaling, unfolded protein response and apoptosis.

Presentation

The SGLT2 inhibitor ertugliflozin causes a switch of cardiac substrate utilization leading to reduced cardiac mTOR-signaling, unfolded protein response and apoptosis.

Empagliflozin improves left ventricular diastolic function in db/db mice without altering cardiac expression of enzymes relevant for ketone body or branched chain amino acid catabolism.

Presentation

Empagliflozin improves left ventricular diastolic function in db/db mice without altering cardiac expression of enzymes relevant for ketone body or branched chain amino acid catabolism.

Myocardial infarction is sufficient to increase GLP-1 secretion leading to improved left ventricular contractility and mitochondrial respiratory capacity

Presentation

Myocardial infarction is sufficient to increase GLP-1 secretion leading to improved left ventricular contractility and mitochondrial respiratory capacity

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