Associate Professor Alessandro Cannavo

Associate Professor Alessandro Cannavo

Federico II University Hospital, Naples (Italy)

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After completing his PhD at Federico II University of Naples, Dr. Cannavo (Ph.D.) worked as a Post-doctoral fellow (from 2012 to 2016) at the Center for Translational Medicine at Temple University (Philadelphia). In 2016 Dr. Cannavo has been awarded with a Postdoctoral Fellowship Grant by the American Heart Association. In recognition of his achievements, in 2016 he has also been awarded of the prestigious Certificate of Research Excellence by the American Heart Association. In 2017, Dr. Cannavo has been named Adjunct Assistant Professor at Temple University. In 2018 He received the prestigious Award from Ministry for Education, University and Research “Rita Levi Montalcini” becoming an Assistant Professor (with Tenure Track, RTDb) at Federico II University of Naples. His publication output enlists 47 peer-reviewed papers in top-tier journals, such as: Circulation (IF 23), Circ Res (IF 15.8), J Am Coll Cardiol (18.6), and Nat Communications (11.8).

Presentations

Deleting cardiac GRK5 catalytic activity impairs basal cardiac function without affecting myocardial growth

Event: Heart Failure 2018

Topic: Cardiac Biology and Physiology

Session: Moderated Poster session 4 - Basic Science

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Myocyte BDNF generation prevents chronic post-ischemic decompensation via cardiac reinnervation: the role of beta-AR and GRK2 signals.

Event: Heart Failure 2018

Topic: Pathophysiology and Mechanisms

Session: Young Investigator Award: Basic Science and Translational Science

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Metoprolol induces cardiac beta-3 adrenergic receptor and Sphingosine 1 phosphate receptor 1 signals to prevent adverse Left-ventricle remodeling and dysfunction after myocardial infarction

Event: Frontiers in CardioVascular Biology 2016

Topic: Signaling

Session: The role of microdomains in beta-adrenoreceptor signalling

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Modulation of catecholamine secretion from human adrenal chromaffin cells by manipulation of G protein-coupled receptor kinase-2 activity.

Event: Frontiers in CardioVascular Biology 2016

Topic: Neurohormones

Session: Growth factors and neurohormones - Heart

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Associate Professor Alessandro Cannavo

Biography

Contributor content

Deleting cardiac GRK5 catalytic activity impairs basal cardiac function without affecting myocardial growth

Myocyte BDNF generation prevents chronic post-ischemic decompensation via cardiac reinnervation: the role of beta-AR and GRK2 signals.

Metoprolol induces cardiac beta-3 adrenergic receptor and Sphingosine 1 phosphate receptor 1 signals to prevent adverse Left-ventricle remodeling and dysfunction after myocardial infarction

Modulation of catecholamine secretion from human adrenal chromaffin cells by manipulation of G protein-coupled receptor kinase-2 activity.

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