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Doctor Lili Xu

Zhongshan Hospital Fudan University, Shanghai (China)
Membership: ESC Professional Member
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Indobufen-based dual antiplatelet therapy in patients with multivessel coronary disease undergoing drug-eluting stent implantation: insight from the OPTION trial
Presentation
Indobufen-based dual antiplatelet therapy in patients with multivessel coronary disease undergoing drug-eluting stent implantation: insight from the OPTION trial
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Dapagliflozin decreases atherosclerotic plaque instability via regulating macrophage pyroptosis
Presentation
Dapagliflozin decreases atherosclerotic plaque instability via regulating macrophage pyroptosis
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Dapagliflozin decreases atherosclerotic plaque instability via regulating macrophage pyroptosis
Presentation
Dapagliflozin decreases atherosclerotic plaque instability via regulating macrophage pyroptosis
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11-year follow-up outcomes of catheter ablation of para-hisian accessory pathways
Presentation
11-year follow-up outcomes of catheter ablation of para-hisian accessory pathways
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Histamine promotes atherosclerotic plaque instability via STAT6-dependent macrophage pyroptosis
Presentation
Histamine promotes atherosclerotic plaque instability via STAT6-dependent macrophage pyroptosis
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Stat4 deficiency exacerbates atherosclerosis through promoting foam cell formation via pi3k/akt/nfkb/mir-9/acat1 pathway in ApoE-/- mice
Presentation
Stat4 deficiency exacerbates atherosclerosis through promoting foam cell formation via pi3k/akt/nfkb/mir-9/acat1 pathway in ApoE-/- mice
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Histamine promotes the differentiation of macrophages from CD11b+ myeloid cells and formation of foam cells through a STAT6-dependent pathway
Presentation
Histamine promotes the differentiation of macrophages from CD11b+ myeloid cells and formation of foam cells through a STAT6-dependent pathway
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STAT4 deficiency exacerbates atherosclerosis by promoting mobilization of myeloid cells, polarization of M1 macrophages and formation of foam cells
Presentation
STAT4 deficiency exacerbates atherosclerosis by promoting mobilization of myeloid cells, polarization of M1 macrophages and formation of foam cells
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