Doctor Preston Mason
Brigham and Women's Hospital, Harvard Medical School, Boston (United States of America)
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ESC Professional Member
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Biography
Dr. R. Preston Mason is a faculty member of the Cardiovascular Division at Brigham and Women’s Hospital and Harvard Medical School in Boston, Massachusetts, USA. Dr Mason is also President and Founder of Elucida Research. He has received many awards for his research in cardiovascular pharmacology and teaching over the past 30 years, including an honorary doctorate in science. He received a full scholarship for graduate and medical programs (MD/PhD) at the University of Connecticut School of Medicine. Prior to coming to Boston, he was on the faculties of the University of Connecticut School of Medicine and the Medical College of Pennsylvania (now Drexel University) where he was nominated for full professor. Dr. Mason has authored or co-authored more than 250 scientific articles and abstracts.
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![Eicosapentaenoic acid (EPA) inhibited lipoprotein(a) [Lp(a)] oxidation and its effects on expression of oxidative stress and pro-inflammatory proteins in endothelial cells](https://cf-images.us-east-1.prod.boltdns.net/v1/jit/6169276219001/9b0201f2-23d2-4043-9b5b-e0a92e376e5d/main/1280x720/2m358ms/match/image.jpg)
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Eicosapentaenoic acid (EPA) inhibited lipoprotein(a) [Lp(a)] oxidation and its effects on expression of oxidative stress and pro-inflammatory proteins in endothelial cells
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Atorvastatin active metabolite inhibits oxidation of Lp(a), small dense LDL and triglyceride-rich lipoproteins compared to other statins through a free radical scavenging mechanism
![High glucose enhanced lipoprotein(a) [Lp(a)] oxidation in a manner inhibited by eicosapentaenoic acid (EPA) in vitro](https://d1ukaf53hjlapf.cloudfront.net/pdf-thumbnails/thumbnail_285997.png)
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High glucose enhanced lipoprotein(a) [Lp(a)] oxidation in a manner inhibited by eicosapentaenoic acid (EPA) in vitro
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Omega-3 fatty acids differentially reduced expression of neutrophil degranulation-associated proteins in endothelial cells during IL-6 exposure
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Eicosapentaenoic acid inhibits lipopolysaccharide (LPS)-induced nitrite production and cytokine release from J774 macrophages
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Omega-3 fatty acids differentially alter the expression of detoxification enzymes and nitric oxide bioavailability in endothelial cells during IL-6 exposure
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