In order to bring you the best possible user experience, this site uses Javascript. If you are seeing this message, it is likely that the Javascript option in your browser is disabled. For optimal viewing of this site, please ensure that Javascript is enabled for your browser.


This content is currently on FREE ACCESS, enjoy another 64 days of free consultation

 

Cardiac arrest predictors in cardiogenic shock

Session Poster Session 3

Speaker Helder Santos

Congress : Heart Failure 2019

  • Topic : coronary artery disease, acute coronary syndromes, acute cardiac care
  • Sub-topic : Coronary Artery Disease: Treatment, Revascularization
  • Session type : Poster Session
  • FP Number : P1797

Authors : H Santos (Barreiro,PT), T Vieira (Porto,PT), J Fernandes (Porto,PT), R Pinto (Porto,PT), T Filipa (Porto,PT), AR Ferreira (Porto,PT), M Rios (Porto,PT), T Honrado (Porto,PT)

15 views

Authors:
H Santos1 , T Vieira2 , J Fernandes2 , R Pinto3 , T Filipa3 , AR Ferreira2 , M Rios2 , T Honrado2 , 1Hospital N.S. Rosario, Cardiology - Barreiro - Portugal , 2Sao Joao Hospital, Intensive Care - Porto - Portugal , 3Sao Joao Hospital, Cardiology - Porto - Portugal ,

Citation:

Background: Cardiac arrest (CA) is a defining moment to patient outcome. Cardiogenic shock (CS) is the cardiovascular emergency with more mortality rates, and CA is considerably more frequent in these patients. Combined, CS and CA, are a potentially lethal event.

Purpose: Evaluate the impact of cardiovascular previous history, clinical signs and diagnosis procedures at admission in the prediction of CA in CS patients.

Methods: Single-centre retrospective study, engaging patients hospitalized for CS between 1/01/2014-31/10/2018. Epidemiological, clinical data at admission and diagnosis procedure’s results were collected. Chi-square, Fisher and T-student tests were used to compare categorical and continuous variables. Logistic regression was performed to assess CA occurrence based on the cardiovascular history, clinical signs and diagnostic tools at admission.

Results: 218 patients were included, mean age 62.34±13.90 years, with 68% males. Patients that suffer CA (134 patients) were similar regarding age, gender, body mass index, arterial hypertension, dyslipidemia, previous ACS and arterial pressure at admission. Active smoking status was more frequent in CA (47 vs 32.1%, p=0.03), on other hand diabetes (20.1 vs 34.5%, p=0.018) and previous cardiomyopathy (23.1 vs 38.1%, p=0.018) were less prevalent that in non-CA patients. Beta blockers (43.9 vs 23.9%, p=0.002), angiotensin-converting-enzyme inhibitor (50.6 vs 34.6, p=0.021), furosemide (25.6 vs 13.4%, p=0.018) and platelet antiaggregants (41.5 vs 23.1%, p=0.004) are more frequent in non-CA patients. CA patients with CS had at admission lower values of pH (7.21±0.23 vs 7.39±0.10, p=0.009), however, higher lactate levels (6.5±6.45 vs 4.7±4.12, p?0.001) and de novo cardiac arrhythmias (23.1 vs 4.8, p?0.001). CA patients had less time until angiography performance (3.0±2.5 vs 5.0±4.5 hours, p?0.001), but better left ventricular ejection fraction (LVEF) (38.11±13.69 vs 25.43±6.43%, p=0.001). Logistic regression revealed LVEF (odds ratio (OR) 1.04, p=0.001, confidence interval (CI) 1.015-1.064) and de novo arrhythmia (OR 8.248, p=0.008, CI 2.38-28.63) as predictors of CA in CS patients.

Conclusions: LVEF and de novo arrhythmia at admission in CS were significant predictors of CA arrest.



Based on your interests

Three reasons why you should become a member

Become a member now
  • 1Access your congress resources all year-round on the New ESC 365
  • 2Get a discount on your next congress registration
  • 3Continue your professional development with free access to educational tools
Become a member now

Our sponsors

ESC 365 is supported by Bayer, Boehringer Ingelheim, Bristol-Myers Squibb and Pfizer Alliance, and Novartis Pharma AG. The sponsors were not involved in the development of this platform and had no influence on its content.

logo esc

Our mission: To reduce the burden of cardiovascular disease

Who we are